by Tomczak, M.F. et al.
- Text changed:
The defect in response to IL-10-Ig appeared to be primarily the result of the absence of thep50
/p105
subunit, because the ability of IL-10-Ig to inhibitcolitis
was also compromised inp50
(-/-); RAG-2(-/-) (p50
/RAG) mice[Ref]
.